Dermatology

Skin inflammation and extracellular matrix modulation

Modulation of inflammatory mediators and extracellular-matrix maintenance in skin cells.

Biological pathway

Reduced pro-inflammatory signalling alongside effects on collagen, elastin and other extracellular-matrix components.

Evidence summary

Laboratory and early cosmetic clinical evidence has been reported for acetyl dipeptide-31 amide, but independent replication is limited.

Limitations

Available studies are small, product-associated and do not isolate all formulation components.

Peptides linked to this mechanism

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